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One curious characteristic of the Spitz tumor pathway is inactivation of CDKN2A (often the only genetic alteration), with loss of p16 expression, which would be of concern in melanocytic tumors in any other pathway.47 Therefore, malignant transformation requires other mutations in addition to inactivation of CDKN2A, for example, TERT promoter mutations

We hypothesize that the role of HPV-16 E7 is to establish a subset of GSTP1 molecules that is inaccessible to oxidative attack, thus creating a reservoir of reduced monomeric GSTP1

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Mental health overlap