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231 Similarly, exogenous supplementation with inhibitor of differentiation-2 (ID2) increases the level of Muc2 mRNA and the expression of Claudin-1 and ZO-1 by inhibiting the activation of neutrophil NF-kB

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Numerous studies have verified the role of the HGF/c-MET pathway in sustaining an immunosuppressive TME, as it obstructs CTL activation and promotes CTL exhaustion (Fig