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Once the growth plates in a child's bones have fused, HGH stops working in this way

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GLP-1R activation exerted antifibrotic effects by blocking the interaction between the NLPR3 inflammasome and PFKFB3-driven glycolysis, leading to the inhibition of lactate production and subsequent histone lactylation, and thereby decreasing profibrotic gene expression in fibroblasts under inflammatory stress

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