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The fact that nrc2 , a mutant affected in GSH2, also exhibited greater sensitivity in its response to AITC confirmed that the root length phenotype was due to the impairment in the glutathione biosynthetic pathway, irrespective of whether GSH1 or GSH2 carried the mutation

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BPC-157 accelerates the healing of injured ligaments and tendons in animal models by promoting fibroblast migration and angiogenesis. Sikiric et al., Pharmacological Research 2