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A biallelic mutation (E366G) in SNIP1 , located near the FHA domain, has been associated with a complex neurodevelopmental disorder in 35 individuals from the Amish community 47,48 , although the underlying mechanistic defect caused by the mutation is unknown

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doi: 10.1371/journal.pcbi.1010408 30 DenkoNC

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Together with GHK-Cus ability to improve vascular wall repair and reduce oxidative stress, this trio supports nervous system recovery, cognitive function, and peripheral circulation integrity in experimental models