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The DLS data of particles showing average size 119.1 nm exceeding the value from AFM data (Fig

In the tumor microenvironment, this metabolic adaptation likely enables tumor cells to continue growing under adverse conditions, suggesting that glutamine metabolism may serve as a critical compensatory mechanism in iron-deficient states

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486 Overexpression of GPX4 in cardiomyocytes or targeting the mitochondrial DOX-Fe 2+ complex prevents DOX-induced ferroptosis and improves cardiomyocyte injury

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