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Similarly, nanoparticle-induced ROS can activate NLRP3 inflammasomes, enhancing phagocytic clearance of pathogens

Given the neuroprotective potential of intact BDNF/TrkB signaling, therapeutic strategies aimed at augmenting this pathway are of significant interest, mirroring approaches explored in other neurodegenerative contexts like Huntingtons disease (Couly et al., 2021)

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However, a surprising discovery revealed that cellular senescence may elicit stemness, and that this mechanism plays an important role in cancer relapse [ 4 , 5 ]