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By supporting barrier integrity and reducing inflammation, these peptides may decrease autoimmune triggers originating in the gut

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There is a bidirectional link between the two phenomena: an increase in permeability promotes dysbacteriosis ( + (CD4 + ) T-cell expansion and interleukin 17A production that limits enteric pathogen invasion ( Salmonella typhimurium infection is more severe in transgenic mice with increased intestinal permeability, suggesting that barrier defects ultimately result in enhanced disease progression despite the activation of protective mucosal immunity ( Intestinal mucus is a critical component of the IEB since it forms a direct link between the host and the microbiota

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The right dose can be the difference between a life-changing treatment and a frustrating experience

BPC-157 has been a game changer for managing injuries