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Paeoniflorin activates the AMPK/SIRT1/PGC-1 axis to mitigate mitochondrial dysfunction, while atractylenolide III upregulates antioxidant enzymes and activates the PI3K/Akt/mTOR pathway to inhibit excessive autophagy (b)

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Impairment of Nrf2/ARE signaling has been observed in HCV infection, leading to elevated ROS levels that can activate autophagy and facilitate HCV maturation and release 103,104

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This depletes our physiological resilience and metabolic reserve, and can lead to malfunction [1]

This does not mean the science is irrelevant