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Although hydrolyzed FB1 (HFB1) is not toxic, the C2 amino group can be acylated by fatty acids in vivo to generate a ceramide synthase inhibitor that is 10-times more toxic than FB1 in mammalian HT-29 cells (Seiferlein et al., 2007)

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(2002) demonstrated that the inability to detect ATP-dependent GSH transport in previous studies with mammalian plasma membrane vesicles was due in part to the inhibitory effect of dithiothreitol (DTT) on Mrp2-mediated transport

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10.1016/j.niox.2008.08.003 15