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The current analysis of the risk factors for bone graft infection after cranioplasty

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Neutrophils are signaled to the areas of inflammation even without infection present, but bacterial presence can push this neutrophilic response further, generating more oxidative stress from released reactive oxygen species, more crosslinking of the mucins from the oxidative stress, and more DNA released from dying epithelial and neutrophil cells (Yuan et al., 2015

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doi: 10.1111/j.1346-8138.2001.tb00134.x

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