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In these reports, elevated tCho has been interpreted as evidence of membrane or myelin disruption, increased membrane turnover, neuroinflammation, or increased cell density

Personalized prediction of the secondary oocytes number after ovarian stimulation: a machine learning model based on clinical and genetic data

Understanding your genetic predisposition to IBS-related traitssuch as visceral sensitivity or transit-time regulationallows PlexusDx clinicians to tailor your dose escalation and compound selection

Smad2/3 form a complex with Smad4, which is also a co-mediator of the bone morphogenic protein (BMP) signaling pathway

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The additional metabolic stress and inflammation associated with obesity can worsen the underlying pathophysiology of lipedema, leading to faster disease advancement