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Therefore, we will review the role of ROS-induced lipid peroxidation in chondrocytes and its decomposition end products 4-HNE and MDA in OA

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Because results from prospective trials are limited by low statistical power, and because retrospective studies are often subject to bias, a pooled analysis of phase III clinical trials and two endpoint trials was performed

Mechanistically, these effects are mediated in part through TSH receptor signaling in hepatocytes and HSCs, which promotes inflammatory and fibrogenic responses (108)

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