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In many cases they possess an N-terminal signal peptide mediating correct subcellular sorting and trafficking and an anionic propeptide which is thought to be the major contributing factor inhibiting the antimicrobial activity by neutralizing the net positive charge of the mature peptide [19, 20]

By disrupting this interaction, the peptide releases p53 to trigger apoptosis selectively in senescent cells while sparing healthy cells (Baar et al., 2017)

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Pentadecapeptide BPC 157 Resolves Pringle Maneuver in Rats, Both Ischemia and Reperfusion

Their complementary mechanisms make them a valuable set of tools for research in regenerative medicine

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