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SFAs exacerbate inflammation via c-Jun N-terminal kinase (JNK)/IKK/NF-B activation (JNK/IKK/NF-B signaling pathway), promote diacylglycerol (DAG) accumulation and protein kinase C (PKC) activation (inhibiting insulin receptor substrate (IRS) phosphorylation), and generate Cer that induces Akt dephosphorylation and oxidative stress

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