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In the present study, autophagy was induced by high glucose concentrations, as directly observed by the accumulation of autophagic vacuoles in the cytoplasm, as well as by the increase in the content of the autophagic markers, LC3-II and Beclin1
It is becoming more widely acknowledged that ferroptosis, a newly identified kind of controlled cell death triggered by iron-dependent lipid peroxidation, plays a major role in the pathophysiology of ARDS ( Figure 2 )
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