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OSA is associated with serious downstream risks, including hypertension, cardiovascular disease, type 2 diabetes, stroke, and cognitive decline, making early and effective treatment critical

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Previous studies have reported that ferroptosis is regulated by multiple metabolic and signaling pathways, and we summarized the roles of these pathways in ferroptosis

(4) After cell stress or injury, these cells are posited to activate innate immune pathways, especially Toll-like receptor (TLR) signaling, which may drive inflammatory responses inside the cell

[3] Because MK-677 activates the ghrelin receptor, it is accurately described as a ghrelin receptor agonist or ghrelin mimetic. At the cellular level, GHSR1a is a Gq-coupled receptor: activation drives phospholipase C, inositol trisphosphate, and a rise in intracellular calcium in pituitary somatotrophs, which triggers GH release