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Beyond glucose control, both treatments improve multiple cardiometabolic risk factors

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In contrast, a number of studies have noted AngIV and AngIV analog-induced facilitation of memory consolidation, retrieval and long-term potentiation (LTP), a phenomenon considered to be the building block of memory [48]

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Moreover, tetracyclines regulate inducible NO synthase (iNOS) at the post-transcriptional level, thereby decreasing NO levels in LPS-stimulated murine macrophages [36, 37, 43, 44]