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However, in female hepatic ER knockout mice fed a high-fat diet, disrupted liver AA homeostasis and significant liver lipid accumulation were observed (80)

albicans cells and its binding to intercellular targets was proposed as a mechanism of yeast inactivation, before subsequent perturbation of the cell membrane (Shagaghi et al., 2017)

For children and adolescents, obesity prevention and management should prioritize non-pharmacological interventions

In addition to GPX4, other regulatory mechanisms have recently been discovered controlling the lipophilic redox balance such as ferroptosis suppressor protein 1 (FSP1) 17,18 and GTP cyclohydrolase-1 (GCH1) 19

Next, the effects of SR4 on liver oxidative stress and GSH-linked antioxidant system were examined

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