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This was observed in genetic models of hypertension, such as aged spontaneously hypertensive rats, 111, 112 stroke-prone spontaneously hypertensive rats, 113 Type 2 diabetic, mildly hypertensive GotoKakizaki rats 114 and TGR(mREN2)27 renin transgenic rats, 115 as well as in several models of drug-induced hypertension

Understanding the molecular and cellular mechanisms underlying the loss or gain of disorder-related detrimental effects mediated by astrocytes in different contexts has the potential to identify new treatment strategies for a wide variety of CNS disorders

The cells were maintained in an incubator at 37 C, with above 95% humidity and 5% CO 2