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Additionally, it serves as a signaling mechanism, influencing cellular pathways, such as cell cycle control, histone ubiquitination, and DNA repair [52]

At the molecular level, hepatic Gclc or Gclm KO triggers a robust activation of Nrf2 and its target genes in liver (12, 13), indicating increased oxidative stress response, whereas Nrf2 level is not increased in livers with Chac1 overexpression

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LWT 164:113634

doi: 10.1111/febs.14740 54 LebeaupinCValleDHazariYHetzCChevetEBailly-MaitreB