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The compound exerts dual benefits in neuropathic mice, normalizing depression-like behaviors through supraspinal serotonergic modulation (enhancing 5-HT signaling) and GABAA receptor activation while simultaneously reducing neuropathic pain symptoms (Zhao et al., 2014)

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4-hydroxynonenal self-limits Fas-mediated DISC-independent apoptosis by promoting export of Daxx from the nucleus to the cytosol and its binding to Fas

However, the mechanism(s) of how mutations in the VKORC1 gene mediate insensitivity to coumarins in vivo remains to be elucidated

Metabolic profiling indicates impaired pyruvate dehydrogenase function in myalgic encephalopathy/chronic fatigue syndrome

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