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A current paradigm in longevity research is that nucleocytoplasmic NAD + levels decline with aging in one or more key tissues and restoring NAD + levels leads to lifespan extension

Afterwards, excessive ROS would interfere with Ca 2+ homeostasis in cardiomyocytes, affecting multiple ion transporters responsible for myocardial electrical activity and bringing about diastolic and systolic dysfunction and arrhythmia

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Mitochondrial complex II: at the crossroads

Differential binding of compounds to the DBD of FOXO isoforms based on subtle differences between these domains and their interaction with the consensus DNA sites could also be exploited

Since then, it has become a key area of interest in life science research