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Studies demonstrate glucagon receptor stimulation increases oxygen consumption and carbon dioxide production, reflecting elevated metabolic rate

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Previous research on DGAT1 -/- mice demonstrated that DGAT1 was required for hepatic steatosis caused by a high-fat diet or fasting, both of which promote hepatic uptake of exogenous FAs, but not for hepatic steatosis caused by upregulation of endogenous de novo FA synthesis 65

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