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doi: 10.1016/j.kint.2017.02.011 89

FIGURE 2 FIGURE 3 The sym33-2 mutant manifested a severe phenotype characterized by the formation of a highly branched network of locked infection threads without bacterial release that are arrested in the root outer cortex

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BPC-157 upregulates growth hormone receptor expression in tendon fibroblasts and activates the JAK2 signaling pathway, directly enhancing fibroblast proliferation and collagen synthesis, according to a study published in Molecules

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