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Administration of pharmacological levels of GLP-1 analogues resistant to DPP-4, not only increases insulin secretion while inhibiting glucagon release in a glucose-dependent fashion, but also delays gastric emptying and suppresses food intake [1][3]
Although it is not included in the diagnostic criteria of PCOS, there is a crucial role of this metabolic impairment, which along with hormonal abnormalities, increase each other in a vicious circle of PCOS pathogenesis
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Copenhagen (Denmark): European Observatory on Health Systems and Policies
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In vivo Animals and housing All animal procedures undertaken were approved by the local ethics committee and conformed to Home Office regulations
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