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Molecular mimicry is a well-documented mechanism: infections like EBV, Klebsiella, Proteus, and Yersinia share structural homology with human tissue proteins, and the immune response originally directed at the pathogen begins cross-reacting with self-tissue

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Down-regulated peroxisome proliferator-activated receptor (PPAR) in lung epithelial cells promotes a PPAR agonist-reversible proinflammatory phenotype in chronic obstructive pulmonary disease (COPD)

Electrophoretic and kinetic studies of glucosephosphate isomerase (GPI) in two different Japanese families with GPI deficiency