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The main reason is that GSH-Au NCs@His does not have the capacity to form an Au-S bond, which greatly reduces the effectiveness of GSH-Au NCs@His to consume GSH, thus protecting tumor cells from ROS oxidative damage (Figure 7C)

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625 RNAi-mediated screening revealed that ALOX5 is the primary factor required for ACSL4-independent ferroptosis induced by mutant huntingtin protein HTTQ94

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Herein, rhodamine B-prelabelled sibrotuzumab functioned as a cell filter to pick out the FAP-positive cells in a mixture of HT-1080 wild type cells