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These metabolic disturbances activate several pathogenic pathways that contribute to neuronal dysfunction and death, ultimately manifesting as DPN

Transient transfection of cytosolic and mitochondrial Apollo-NADP + sensors was detected by fluorescence microscopy 48 h post-nucleofection (Fig 4A)

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These results suggest that individuals with certain metabolic states may experience more severe autism, especially as relates to its social manifestations as well as developmental delays

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