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Even when baseline metabolic risks are considered as confounders, these results emphasize that periconception management must look beyond the cessation node and focus on whether weight and metabolism enter a controllable stability window post-discontinuation

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Moylan, Alisa Pedrana, Paula Petrone, Jrn M

These findings suggest that coordinated incretin and glucagon receptor activation may influence pathways involved in hepatic lipid handling and metabolic signaling [5][8]

The study was registered in the EU Clinical Trials Register (EudraCT 2019-000781-38)

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