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Interestingly, EMPA exhibited an antiepileptic effect against PTZ-induced seizures through significantly reducing neuronal PAS domain Protein 4 (Npas4), cyclic adenosine monophosphate (cAMP) response element binding protein (CREB) hippocampal expressions, and enhancing the brain-derived neurotrophic factor (BDNF)-tropomyosin receptor kinase B (TrkB) pathway, which are found to be involved in epileptogenesis, eventually leading to significant improvement of behavioral impairments induced by PTZ

AOD-9604 failed Phase 2 not due to adverse events but because the mean fat loss versus placebo, while statistically significant, was too modest for a standalone obesity treatment

GHK-Cu attracts immune cells (macrophages, mast cells) and endothelial cells to injury sites, stimulates angiogenesis (new blood vessel formation), and promotes nerve outgrowth

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Urolithin A alleviates acute kidney injury induced by renal ischemia reperfusion through the p62-Keap1-Nrf2 signaling pathway

T., Filonenko, V