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Essentially, the peptide acts as a biological switch that initiates self-sustaining healing programs rather than requiring continuous presence [23, 26, 39, 54]

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In contrast, vitamin K antagonists, exemplified by warfarin, could induce ferroptotic damage within RPE cells, promote the related vascular proliferation, and ultimately, exacerbate CNV lesions, a sequence of events associated with more pronounced and expeditious vision loss in AMD (148)

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J., Vieth, R., Umhau, J