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In the field of delivery technology, targeting the unique joint anatomy and pathological environment of OA, developing biodegradable sustained-release carriers such as smart responsive nanogels, porous microspheres, or tissue engineering scaffolds is key to achieving long-term and controlled release of GPX4 genes, regulatory miRNAs or ASOs, or small molecule ferroptosis inhibitors at the lesion site ( Considering the multifactorial pathogenesis of OA, future research needs to prioritize conducting early proof-of-concept clinical trials to evaluate the feasibility and synergistic effects of locally applied ferroptosis inhibitors such as Fer-1 or GPX4 activators combined with existing conservative treatments including weight management, physical therapy, and NSAIDs or emerging cell and tissue engineering therapies
These are capable of creating OS [3], which is caused by an increased level of ROS or decreased level of antioxidants
BIBLIOGRAPHIE A Garrido Pertierra
Dean OM, van den Buuse M, Berk M, Copolov DL, Mavros C, Bush AI
dr- [email protected] (A..) 3 Department of Anesthesiology and Reanimation, Faculty of Medicine, Gazi University, Ankara, Turkey
