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Altered B12 handling and reduced clearance in kidney disease, and both cellular release and reduced clearance in liver disease, can raise B12, so supportive renal or hepatic findings make those causes plausible

In atherosclerotic cells, such as ox-LDL-treated macrophages, copper levels are significantly elevated (by approximately 62%), providing a catalyst for redox reactions [154]

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Dewey KG et al

After 3 weeks I havent noticed any changes at all

The 200 to 500mcg figures widely used across clinic and community contexts are derived from animal extrapolation and accumulated self-experimentation, not controlled human trials