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Novo Nordisks Peter Johannsen, MD, highlighted the biological signals observed and acknowledged the clinical limitations: subgroup analyses did not reveal meaningful benefit, and any potential responder group would be very small

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Furthermore, other pathophysiological abnormalities [5], such as increased renal glucose re-absorption [6] and the diminished incretin effect [7] have been found to contribute to the hyperglycemia manifested by the disease

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