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Likewise, its easy applicability during the course of an injurys treatment might bypass the limitation of the standard agents (for review, see [1,3,4,5,6,8,9,32,33,34,92]) (i.e., the greater effect normally thought to result from prevention than treatment [35,36,37,38,39,40,41,42])

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Regarding histone acetylation, NADPH oxidase plays a critical role in producing lipid ROS, and inhibitors of histone deacetylases can inhibit the accumulation of lipid ROS by suppressing the expression of NADPH oxidase genes [60, 73]

Characterization of human glutaredoxin 2 as iron-sulfur protein: a possible role as redox sensor