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The mRNA and protein levels of the hub genes decrease in a dose-dependent manner in FIN56-treated GC cells, indicating that FIN56 regulates hub gene expression during ferroptosis and suggesting its potential as a drug for inhibiting invasion by GC cells [91]

Following ingestion, PBAT-MPs accumulate preferentially in intestinal mucosa, where they induce gut dysbiosis characterized by reduced microbial diversity and altered metabolite profiles [10]

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However, researchers undertaking long-term GHK-Cu studies or testing high-amount dosage protocols are advised to monitor subjects total copper levels