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Therefore, the evidence of controlling both the egr-1 gene and its repressor nab2 gene through BPC 157 therapy [100] provides the basis for the belief that with BPC 157, uncontrolled egr-1 gene expression would not occur

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As noted, BPC 157 therapy acting via NO-system distinctive presentations previously specified (i.e., L-NAME-responsible/L-arginine responsible, L-NAME-non-responsible/L-arginine-responsible) counteracted opposite disturbances (catalepsy (haloperidol) vs

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