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Indeed, in reporting our study, we argued that the degree of cell proliferation seen prior to embryonic loss in TrxR1-deficient embryos was inconsistent with a block to proliferation, and instead suggested that the pre-resorption phenotype of TrxR1-deficient embryos was more consistent with an embryonic patterning defect (Bondareva et al., 2007)(see below)

Body weight in the semaglutide group decreased by an estimated 6.8 kg more than placebo, confirming the expected weight loss effect, but the bone changes occurred in parallel rather than being fully explained by mechanical unloading from weight loss alone