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The preclinical evidence is robust, consistent, and mechanistically plausible, but the gap between rat sciatic nerve transection models and human diabetic neuropathy is significant

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Finally, for therapy effect of BPC 157 on paracetamol brain injury in rats that received pentadecapeptide BPC 157 later in an advanced paracetamol toxicity stage, we should consider that at the time point of 3 hours following paracetamol, as previously mentioned, after therapy these rats had initially exhibited generalized convulsions and had significant brain damage

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