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For the L-GSH supplementation group, the levels of reduced form of GSH remained relatively the same and were not significantly different between pre- and post- L-GSH supplementation in plasma ( Figure 4F ) and PBMCs ( Figure 4G )

Beyond glucose lowering, incretin-based therapies are increasingly positioned within the broader cardiometabolic framework, with emerging data supporting cardiovascular and renal protection, as well as effects on endothelial function, atherosclerosis, heart failure phenotypes, and chronic kidney disease progression

Cholesterol efflux from macrophages to HDL plays an important role in protecting from the development of atherosclerosis