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Within this pathophysiological context, SIRT1 acts as a central regulator at the interface of metabolic, inflammatory, and oxidative processes, contributing to hepatic lipid reprogramming, insulin sensitivity, ROS management, autophagy maintenance, and gutliver barrier protection

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In an open controlled trial of 60 patients with chronic lymphocytic thyroiditis (59), the combination of L-T4+ Se possessed better efficacy than L-T4 monotherapy compared to the L-T4 group, TPO-Ab and TG-Ab were significantly reduced

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