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doi: 10.3390/jcm13185403

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Furthermore, a Lck C3,5 chimera which was anchored at the plasma membrane using a transmembrane domain instead of site-specific palmitoylation was unable to efficiently reconstitute TCR signaling, indicating that palmitoylation might play an important role in promoting TCR signaling beyond Lck membrane-binding (143)

This non-active form of B12 needs to be converted in the body to an active form, versus the readily active version Methylcobalamin

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