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Indirectly, oncogenic Rho GTPase signaling activates JNK (c-Jun N-terminal kinase), which phosphorylates and stabilizes c-Jun, forming a coherent JNK/cJun/GLS promoter axis that amplifies GLS expression and promotes glutamine metabolism in breast cancer cells (90)

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Furthermore, essential metabolic pathways of bacteria, such as folate biosynthesis and amino acid metabolism, provide potential novel targets for antibiotic development

[see Drug Interactions (7.1)]