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If these clinical trials show limited toxicity, the potential use of MC4R for ASD human research clinical trials may be a prominent step forward as a potential novel neurotherapeutic agent for the treatment of this complex disorder

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thereby, the CREB1-NRF2 signaling pathway potentiates the function of MSCs, which was shown to increase their therapeutic potential in a humanized mouse model of graft-versus-host disease [37]

Two unrelated Chinese patients with hyperinsulinism/hyperammonemia (HI/HA) syndrome due to mutations in glutamate dehydrogenase gene., J Pediatr Endocrinol Metab 2010;23:733-738

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