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These changes result in an overall amplification of intracellular estrogen bioavailability and support a mechanism by which local concentrations of estrogen can change quickly following acute stress or damage (Pomari et al., 2015)

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While studies explore plausible mechanisms (NO-system modulation, VEGFR2-Akt-eNOS signaling, GHR/JAK2 in tendon cells), well-controlled human trials are limited

RESULTS AND DISCUSSION Among the four tested carbon source, glucose supported the maximum growth followed by sucrose