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Recent studies have brought our attention to the mechanisms of glucagon-like peptide 1 (GLP-1)

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Another limitation is the lack of data on the evolution of BMI during the follow-up period, which could affect the interpretation of our results

CVS may be best described as a consequence of dysfunction in the brain stem and hypothalamic nuclei that normally modulate or gate sensory emetic inputs, leading to the failure of brain integration and filtering mechanisms and resulting in the activation of emetic neurocircuitry under normal conditions

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Can I switch from semaglutide to tirzepatide mid-treatment