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Glutathione functions through two primary mechanisms: direct scavenging of reactive oxygen species (ROS) and serving as a cofactor for glutathione peroxidase, a family of enzymes that neutralize hydrogen peroxide and lipid peroxides [2][4]

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Thus, while hypoxia remains one of the strongest predictors of radiotherapy failure, traditional solutions are constrained by unacceptable toxicity risks

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Annu Rev Physiol (2022) 84:43559

HDAC can mediate the shuttling of HMGB1 between the nucleus and the cytoplasm