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Lysosomal dysfunction in the brain of a mouse model with intraneuronal accumulation of carboxyl terminal fragments of the amyloid precursor protein

Alterations of phospholipid metabolites in postmortem brain from patients with Alzheimers disease

Case 2 Investigations: A 43-year-old woman presented with generalized weakness, weight loss, and abdominal pain, prompting a diagnostic workup

genetic deletion or small-molecule inhibition of ALKBH5 reduces these suppressive infiltrates and improves anti-PD-1 efficacy in multiple syngeneic models, directly tying an m 6 A eraser to the composition of tumor-infiltrating immune cells

Phosphorylation at multiple sites has been determined to initiate proapoptotic behaviour of cerebellar granule neurons (Reddy et al., 2013)

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