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Archived from the original on 19 May 2020

EBioMedicine (2019) 50:40820

Journal of Physiology and Pharmacology , 74(2), 231249

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Mitochondrial-mediated ROS cause oxidative damage to cellular structures, particularly mitochondrial DNA, somatic mutations, genome instability, inactivation of tumor suppressor genes, alterations in metabolic and signaling pathways with simultaneous activation of compensatory antioxidant mechanisms, all of which contribute to cell transformation and thus promote various types of cancer [42]